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Nrf2-Heme oxygenase-1 modulates autophagy and inhibits apoptosis triggered by elevated glucose levels in renal tubule cells |
Joo-Heon Kim, Kyeong Min Kim, Jin Uk Jeong, Jong Ho Shin, Jae Min Shin, Ki Tae Bang |
Kidney Res Clin Pract. 2019;38(3):318-325. Published online September 30, 2019 DOI: https://doi.org/10.23876/j.krcp.18.0152 |
Nrf2-Heme oxygenase-1 modulates autophagy and inhibits apoptosis triggered by elevated glucose levels in renal tubule cells MON-207 THE NRF2-HEME OXYGENASE-1 SYSTEM MODULATES AUTOPHAGY AND INHIBITS HIGH GLUCOSE INDUCED APOPTOSIS IN RENAL TUBULAR EPITHELIAL CELLS HEME OXYGENASE-1 VIA TRANSCRIPTIONAL ACTIVATION OF NRF2 ATTENUATES APOPTOSIS IN A549 LUNG CANCER CELLS Nrf2-Heme Oxygenase-1 Attenuates High-Glucose-Induced Epithelial-to-Mesenchymal Transition of Renal Tubule Cells by Inhibiting ROS-Mediated PI3K/Akt/GSK-3β Signaling Ampelopsin Inhibits H2
O2
-induced Apoptosis by ERK and Akt Signaling Pathways and Up-regulation of Heme Oxygenase-1 Retracted: Involvement of Nrf2-Mediated Upregulation of Heme Oxygenase-1 in Mollugin-Induced Growth Inhibition and Apoptosis in Human Oral Cancer Cells Berberine activates Nrf2 nuclear translocation and inhibits apoptosis induced by high glucose in renal tubular epithelial cells through a phosphatidylinositol 3-kinase/Akt-dependent mechanism Netrin-1 alleviates high-glucose-induced renal proximal tubule injury by activating AKT/mTOR-mediated autophagy Simvastatin attenuates oleic acid-induced oxidative stress through CREB-dependent induction of heme oxygenase-1 in renal proximal tubule cells Heme Oxygenase-1 Promotes Survival of Renal Cancer Cells through Modulation of Apoptosis- and Autophagy-regulating Molecules |